CAUSES OF EARLY AND LATE MENOPAUSE, PART II

CAUSES OF EARLY AND LATE MENOPAUSE, PART II

In fact, absolutely everything that we call hormonal regulation is built on this relationship and even the interdependence of the levels of various hormones in the blood. And it is precisely these connections that cause the majority of avalanche-like hormonal disruptions, in which medicine is often not able to determine which gland they started with. The main professional task of any endocrinologist is not to look at the analysis and identify the high / low level of some hormones at the moment. His task is to find the right starting point from which these “races” began. And since there are eight endocrine glands in the body, and they produce a total of more than 100 hormones, it is not as simple as it might seem. Therefore, a typical mistake of an endocrinologist is to prescribe a course of either an antagonist to a hormone that is elevated in the patient, or that hormone that he clearly lacks. Without understanding the true cause of the failure, this all leads to an aggravation of the imbalance. And such an outcome in endocrinology is a very common phenomenon.

We were a little distracted by the story about the difficulties of organizing the endocrine system for a reason. As we can see, a healthy norm here depends on how correctly and stably each of the 8 endocrine glands of the body works. Because one will begin to “junk” – and the rest will be included in the process over the next year. In addition, any hormones, like most substances produced in the body, are proteins. And we are well aware that the principles of building all of our own body proteins are laid down in its genetic code, that is, the set of chromosomes that we get from mom and dad at conception and which is duplicated in every cell of our body.

Simply put, the quality of the endocrine system is associated not only with how well it is formed and how much it is altered by previous diseases. It also depends on how correct in every respect the molecules are produced by each endocrine gland. And of course, from how correct are the principles of regulation that the pituitary gland learned in the process of practical work …

Therefore, if we say that most of the features of the endocrine system are of hereditary origin, this will not seem strange anymore. Indeed, a whole series of phenomena from the category of “not like everyone else” is formed here in our teens. And in the future it is only fixed. And when we turn in search of an answer to a family history, it usually turns out that our grandfathers and grandmothers also had this feature. If we want to find out what kind of deviations our own children will have, we should interview our parents. Indeed, hereditary traits have the property to manifest themselves after 1 generation.

And all this means that, if there are cases of early or late menopause in family history, we have every chance to inherit this trait. In such cases, it should be accepted as inevitable, since modern science is not able to effectively correct genetic defects. We can’t do anything substantial here. And if we can, no matter how we regret it afterwards. However, we are not talking about a defect here – unless, together with the timing of the onset of menopause, we also inherit other, more threatening features of sexual development. We are talking, of course, about diseases such as cancer and others that can also be inherited. Climax, like any strong hormonal “shake”, in itself is very predisposing to the degeneration of some body tissues.

The danger of a malignant tumor (the scientific term is malignancy of cells) during menopause only increases. And if during our inquiries we, among other things, found out about cases of cancer that occurred during menopause or in postmenopausal women, we should also be wary of this scenario. And be wary of every reason. The defect here is not the termination of reproductive activity, but the malignant process that it triggers.

It is because of the serious malignant “grain” laid in menopause that we say that artificially delayed menopause can bring us more trouble than bonuses. After all, if our whole body carries out one genetic program, and the testes or ovaries do a completely different one, such an artificially created difference can provoke many processes. For example, immunity can begin to fight overly active gland cells, taking them for an aggressor. And such phenomena do not always end with their destruction – sometimes this also leads to their malignancy. In particular, in cases where the immune system does not try to block the cell itself, but the product of its manufacture, the hormone. This affects tissues that are often not even related to the reproductive system – they can simply turn out to be cells that accumulate sex hormones. Usually destroying them is not as simple as the cells of the causative gland. And blocking their vital functions by the immune system may well lead to degeneration.

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